EMF Blocking plus EMF Shielding
No matter if you live in an apartment or house or simply would like to ensure your home is free from EMFs There are a variety of methods to reduce exposure. One of the easiest is to restrict your use of electronic devices. https://ctxt.io/2/AACQWf7AFQ can also turn to EMF block paint to prevent EMF radiation from reaching your home. Another way to shield your home against EMF radiation would be to install a shielding canopy for RF. This is a fabric made of net that has EMF shielding. It's used to block EMFs from entering a room. Another option is to have your home fitted with an enclosure that is conductive. They are referred to as Faraday cages.
Numerous studies have demonstrated that the non-ionizing RF EMF produces antiproliferative effects on HCC cells. The mechanism that drives AM RF EMF's anticancer activity in vitro is believed to be based on the downregulation the cancer stem cell. This could explain the long-term effects observed in some patients with advanced HCC. But, the reason for AM EMF's impact on patients with cancer is not evident.
Aspects on the effects of AM RF EMF on HCC tumour growth in vivo were studied in mice. The tumours were divided into three groups. One group did not have exposure RF EMF. The second group was exposed RF EMF at a frequency that is similar to the one used by humans. In the third, they were subjected RF EMF at HCC-specific modulation frequencies. The effect of HCCMF on tumours was compared to that of RCF. The results showed that the cancers treated with HCCMF had significant shrinkage. However, the tumors treated with RCF didn't show evidence of shrinkage in the tumour.
https://postheaven.net/salarydeal66/emf-blocking-and-even-emf-shielding behind tumour-specific AM RF EMF might be based on the fact that tumour cells require Cav3*2 type voltage calcium channels to promote proliferation and down-regulation. AM RF EMF's antiproliferative effect in HCC cells is mediated by CACNA1H which is a protein that mediates tumour-specific Ca2+ influx. The findings suggest that CACNA1H may have broader implications for treatment and diagnosis of many cancers.
The tumors in the controls were never exposed to EMF from radiofrequency, and fed a standard mouse diet. The tumours in those in the HCCMF group were injected with Huh7 cells when they were 5 to 7 weeks old. The tumours were then euthanized after they had a high burden.
The tumours from the three groups also displayed different growth curves. The HCCMF-treated tumors saw a significant reduction in the size of the tumour after 8 weeks. However, the tumors that were treated using RCF did not show any reduction in size. emf blocking was highly significant. The tumors treated with RCF had necrosis, which is common in tumors that have been that are exposed to RCF. The possibility is that this necrosis was due to a lack of oxygen in the larger tumours.
In sum, the results suggest an AM-RF EMF exhibits anticancer effects in vitro and in vivo. A number of studies have proven the fact that AM RF EMF produces measurable shrinkage of tumors in HCC patients. It is possible that AM RF EMF produces these effects because of CACNA1H, a protein involved in tissue-specific Ca2+ influx. In addition, AM RF EMF may exert a sustained effect on the growth of HCC tumors in vivo.