EMF Block Paint and even EMF Shielding
https://ctxt.io/2/AACQZSDcFg if you live in an apartment or a house or just need to ensure that your house is free of electromagnetic fields There are a variety of ways you can reduce exposure. One of the easiest is to reduce the usage of your electronic devices. Learn more could also consider EMF blocker paint to block EMF radiation from reaching your house. Another way to shield your house against EMF radiation is to put up a RF shielding canopy. This is a cloth of net that contains EMF shielding. blocking emf 's used to prevent EMFs from entering a space. Another option is to have your home fitted with a conductive enclosure. These devices are called Faraday cages.
Numerous studies have demonstrated studies have shown that the nonionizing EMF can cause antiproliferative properties in HCC cells. The mechanism that drives AM RF EMF's anticancer activity in vitro is thought to be based on the downregulation the cancer stem cell. This could be the reason for the long-term response seen in certain patients suffering from advanced HCC. However, the reason behind AM EMF's effects on patients with cancer is not evident.
The effects on the effects of AM RF EMFs on HCC tumour growth in vivo were examined in mice. The tumours were classified in three different groups. First, the group that was unaffected to RF EMF. Second group members were exposed RF EMF at frequencies similar to the frequency used by humans. Third group members were exposed to the RF EMF in HCC-specific frequencies. The effect of HCCMF on tumours was compared to that of RCF. The results revealed that tumours treated with HCCMF had significant shrinkage. However, the tumors treated with RCF showed no evidence of tumour shrinkage.
The mechanism behind cancer-specific AM RF EMF may be driven by the fact that cancer cells require Cav3*2 type voltage calcium channels to promote proliferation and down-regulation. AM RF EMF's antiproliferative effect in HCC cells is caused through CACNA1H, a protein which is responsible for the influx of Ca2+ specific to tumours. The results suggest that CACNA1H may have broader implications in the treatment and diagnosis of various cancers.
The tumours in those in the group that were unaffected to RF EMF, and were fed a normal mouse diet. The tumors of HCCMF HCCMF group were treated with Huh7 cells when they were 5 to 7 weeks old. The tumours were then euthanized when they showed excessive burden.
The tumors of the three groups showed different growth curves. The tumours treated with HCCMF had a significant reduction in size of the tumor after 8 weeks. However, the tumors that were treated using RCF didn't show signs of shrinkage. The difference was highly significant. The tumors treated with RCF had necrosis, which is common in tumors that have been that are exposed to RCF. The possibility is that the necrosis was due to a lack of oxygen in the larger tumours.
In summary, the results show the fact that AM EMF has anticancer activity in vitro and in live. A number of studies have proven it is true that AM RF EMF produces measurable tumour shrinkage in HCC patients. It is possible that AM RF EMF causes these effects because of CACNA1H, a protein involved in tissue-specific Ca2+ influx. In addition, AM RF EMF may exert a sustained influence on the development of HCC tumors in the vivo.